Given that the neutrophilic and proteolytic products generated by bronchiectasis (particularly neutrophil elastase [NE]) seem to play a fundamental role in the disease’s genesis and progression, as has been shown in some studies [21,22], efforts have been made to find anti-inflammatory treatments to block this NE and thereby prevent the damage caused by the inflammation itself [23,24]. This evidence concerns the gene ELANE and bronchiectasis.