The protective ability of PK 11195 to counteract the effect of CoCl2-induced mitochondrial damage via TSPO-related processes in non-neuronal (lung cancer) and central (microglial) cell lines included the assessment of: cell viability, oxidative stress, mitochondrial membrane potential depolarization, TSPO protein expression levels, and apoptotic markers. Here, TSPO is linked to lung carcinoma.