If a slowly transforming retrovirus obtains a proto-oncogene from the host genome, it becomes an acutely transforming virus that can induce rapid-onset tumors within a short time after host infection, such as myc in MC29, CMII and OK10 strains of ALV [6], erbB in avian erythroblastosis virus [39], and v-rel in the T-strain of REV [40]. Here, EGFR is linked to infection.