TP53 and atherosclerosis: Atherosclerosis is provoked and aggravated by HCMV via CD36 expression to promote uptake of OxLDL, US28 expression to stimulate migration of SMC, which is highly involved in the pathogenesis of vascular diseases, the suppression of p53 to increase SMC proliferation, UL122 expression to provoke endothelial cell injury through the translocation of heat shock protein 60 (HSP60), and T-cell expansion and accumulation [71,72,73,74,75,76].