MRC1 and Sepsis: The distribution of M2 macrophages labeled with Mrc1/Cd206 and Cd163 in the sepsis with LL-TS group (subset 3 and subset 7) was higher than that in the sepsis group (subset 4 and subset 9), indicating that LL-TS in sepsis could promote the polarization of cardiac M2 macrophages and inhibit cardiac inflammation (Figure 3).