AKT1 and neoplasm: In addition, miR‐29 via targeting VEGF in PI3K/AKT and JAK/STAT signaling pathways in nasopharyngeal carcinoma 5‐8F cells using qRT‐PCR and Western blot showed suppression by increasing invasion, cells growth, migration, tumor cells proliferation, and angiogenesis via decreased phosphorylation levels of AKT, JAK1, STAT1, and STAT3 (Shi et al., 2019).