Thirteen patients had a variant in a DCM gene that was classified as likely pathogenic or pathogenic and four had a variant of uncertain significance (VUS); nine had likely pathogenic truncating variants in TTN, two had pathogenic missense variants in LMNA, two had variants in DSP (one pathogenic truncating variant, one splice donor variant of uncertain significance), one a likely pathogenic missense variant in TNNT2, two missense variants of uncertain significance in MYH7, and another a missense variant of uncertain significance in NEXN (Tables 2A,2B for variant details). This evidence concerns the gene DSP and familial dilated cardiomyopathy.