As shown in Figures 9A–E, ACKR1, LMNB1, MFAP4, NMU, and SEMA3C were upregulated in ectopic endometrial tissues of patients with endometriosis compared with normal endometrium (P < 0.001), which was consistent with the results of bioinformatics analysis. This evidence concerns the gene ACKR1 and endometriosis.