For example, the glycosylation of epidermal growth factor receptor (EGFR) inhibits the abnormal phosphorylation of EGFR in TKI-resistant lung cancer cell line (Yen et al., 2015); the N-glycosylation at N359 ameliorated the hyperphosphorylated and aggregated of tau (Losev et al., 2021). This evidence concerns the gene EGFR and lung carcinoma.