TRPV1 and myocardial infarction: Furthermore, as a form of cross-talk between fibroblasts and other cardiac tissue components, the presence of TRPV1 in cardiac adrenergic afferent fibres enhanced post myocardial infarction fibrosis in the infarct border zone as well as disrupted connexin connectivity and promoted ventricular arrhythmias (Yoshie et al., 2020).