No tier 1 or 2 pathogenic variants were found within the patient's whole genome, or deleterious biallelic/compound heterozygous variants in bronchiectasis‐associated genes CFTR, SCNN1B, SCNN1A, SCNN1G, 42 OMIM primary ciliary dyskinesia genes or 301 gold standard Syscilia genes (Tables S2 and S3), and the patient was considered unsolved by Genomics England. This evidence concerns the gene SCNN1A and primary ciliary dyskinesia.