EVA1A and cancer: Upregulation of miR-103a-3p significantly promoted the migration, invasion and proliferation of Hccl-M3 cells (P < 0.01, Fig. 5), and overexpression of EVA1A significantly suppressed the migration, invasion and proliferation of Hccl-M3 cells (P < 0.001, Fig. 5), indicating that EVA1A attenuates the cancer-promoting roles that miR-103a-3p plays in Hccl-M3 cells.