In addition to using actin as loading control, PINK1 protein expression normalized to mitochondrial content using TOM20 protein expression levels was also higher in normoxia and hyperoxia-exposed MSCs from infants who survived with no/mild BPD compared to MSCs from infants who died or developed moderate/severe BPD (Supplemental Figure E2). This evidence concerns the gene PINK1 and bronchopulmonary dysplasia.