TRPV2 and viral infectious disease: In line with these observations, the viral titers in the brain and the expression levels of HSV‐1 UL30 gene or VSV N gene in various tissues of Trpv2fl/fl mice but not Lyz2‐Cre;Trpv2fl/fl mice were significantly reduced by SKF96365 treatment (Figure 2F,G), indicating that myeloid TRPV2 is the primary target of SKF96365 in mice defense against viral infection and that SKF96365‐mediated inhibition of TRPV2 activity promotes antiviral responses.