TRPV2 and viral infectious disease: Though the exact mechanisms by which homeostatic Ca2+ by TRPV2 mediated the expression of LRMDA and by which LRMDA regulated the tension of cell membrane are currently unclear, these available data have clearly suggested that the TRPV2‐Ca2+‐LRMDA axis promotes viral infections and that targeting the TRPV2‐Ca2+‐LRMDA axis would provide potential therapeutic interventions for the treatment of virus‐caused diseases.