As shown in Figure 2B, we observed that SKF96365 treatment substantially inhibited the replication of H129‐G4 in Trpv2fl/fl BMDCs but not in Lyz2‐Cre;Trpv2fl/fl BMDCs as monitored by the GFP signals (Figure 2B), indicating that TRPV2 is the primary target of SKF96365 in BMDCs and that inhibition of the ion channel activity of TRPV2 by SKF96365 substantially prevents viral infection in BMDCs. Here, TRPV2 is linked to viral infectious disease.