POPDC1 and chronic obstructive pulmonary disease: Loss of either Popdc1 (a.k.a. Bves) or Popdc2 in mice or zebrafish results in arrhythmic and bradycardic phenotypes with high HR variability (Froese et al, 2012; Kirchmaier et al, 2012; Schindler et al, 2016b), while human mutations of POPDC family members cause limb‐girdle muscular dystrophy (LGMD), cardiac arrhythmia, familial atrioventricular (AV) block, and are implicated in long‐QT syndrome and heart failure (Gingold‐Belfer et al, 2011; Tan et al, 2013; Wang et al, 2016; Schindler et al, 2016b; Brand, 2019; Han et al, 2019; Vissing et al, 2019; Indrawati et al, 2020; Rinné et al, 2020).