Exosomal FTO upregulates ABCC10 expression in the recipient cells in a m6A‐dependent manner to promote gefitinib resistance, indicating that exosome‐mediated RNA modification represents another mechanism that promotes gefitinib resistance in NSCLC.[89] DNMT1 (DNA methyltransferase 1) transcripts are enriched in exosomes from ovarian cancer cells, and coincubation with exosomes stimulates DNMT1 protein expression and renders the resistance of host cells to cisplatin. Here, FTO is linked to non-small cell lung carcinoma.