DNMT1 and ovarian cancer: Exosomal FTO upregulates ABCC10 expression in the recipient cells in a m6A‐dependent manner to promote gefitinib resistance, indicating that exosome‐mediated RNA modification represents another mechanism that promotes gefitinib resistance in NSCLC.[89] DNMT1 (DNA methyltransferase 1) transcripts are enriched in exosomes from ovarian cancer cells, and coincubation with exosomes stimulates DNMT1 protein expression and renders the resistance of host cells to cisplatin.