Eleven variants were pathogenic/likely pathogenic (P/LP) as defined in ClinVar (LDLR:7, LPL:2, LIPA:1, and APOE:1; Table S7); 9 CHD patients and 9 controls had 1 of the 7 P/LP variants within LDLR (OR~5.97). This evidence concerns the gene LPL and coronary artery disorder.