We used established myeloid-derived cell line models of neutrophils (differentiated HL-60 cells)35 and MφRAW264.7 to evaluate the impact of TREM1-decoy peptide LP17 and the HuR dimerization inhibitor SRI42127 on cell chemotaxis towards glioblastoma cells in the trans-well assay under conditions that mimic hypoxia (CoCl2 132 uM) in vitro (Figure 5A–E). This evidence concerns the gene ELAVL1 and glioblastoma.