First, we recently reported that de novo EBV infection within atMBCs (TBX21/T-bet+, ITGAX/CD11c+, FCRL4+, FCRL5+) induced elevated expression of inflammatory mediators and – unexpectedly – neuronal lineage genes involved in nervous system development and axon guidance (15). This evidence concerns the gene FCRL5 and Epstein-Barr virus infection.