CD86 and Epstein-Barr virus infection: Interestingly, GC LZ genes (BATF, CD40, CD83, CD86, CD274 (PD-L1), FAS) were more strongly co-expressed with ICAM1 in LCLs than in tonsils (Figure S3D), suggesting a bias toward LZ-like state enrichment in the context of EBV infection.