NFKB1 and synovial sarcoma: In SS, salivary gland epithelial cells can produce proangiogenic factors by activating the VEGFA/TACE/VEGFR2/NF-kB axis [74–76], but VEGFA-regulated TACE is responsible for the soluble-TNF-α release, and the production of TNF-α is closely associated with SS [77].