In gastric cancer, miR-29b-3p was found to inhibit the emigration and colonization of cells by modulating the autophagy-related protein MAZ [39], while in colorectal cancer, overexpression of miR-29b suppresses the epithelial-mesenchymal transition and angiogenesis via disrupting the ETV4-dependent stimulation of the extracellular signal-regulated kinase (ERK) cascade [40]. Here, MAZ is linked to gastric cancer.