The study deployed GSEA to further explore EMID1 functions in LUAD and specified the following as differentially enriched in its high expression phenotype: melanogenesis, basal cell carcinoma, vasoconstriction, glycosaminoglycan biosynthesis of heparin sulfate, Notch signaling pathway, neuroactive ligand-receptor interaction, Hedgehog signaling pathway, ganglioside biosynthesis series, GnRH signaling pathway, and dilated heart myopathy. This evidence concerns the gene EMID1 and basal cell carcinoma.