Although a basal level of phosphorylation of the AKT1/2, ERK1/2, JNK1/2 or p38 MAPK activation sites was seen, indicating that these pathways are active in these cells, no significant and consistent difference were seen with WT Eμ-Myc lymphomas (Figure 3A,B, Supplementary Figure S6B,C). This evidence concerns the gene AKT1 and lymphoma.