Although we cannot rule out that the other kinases we see being phosphorylated play an important role in the phenotype of Eμ-Myc/cRel−/− lymphomas, we decided to focus on potential changes to the AKT, JNK, ERK and p38 MAPK signalling pathways as these constitute highly studied and druggable targets. This evidence concerns the gene AKT1 and lymphoma.