Among these nine pathways including cell cycle, Hippo, MYC, Notch, NRF2, TP53, PI3K, RTK‐RAS, and WNT, the genomic alterations promoting oncogenic activities while inactivating tumor suppressor functions were much more frequently observed in TS‐CB and TS‐CC than in TS‐CA subtypes (Fig. 3D). Here, TP53 is linked to neoplasm.