Unlike the hypertrophy of myocardium caused by insufficient copper levels throughout the body in the pressure load type, copper levels in the DCM are high in the blood throughout the body but low in the myocardium cells [18] that may be related to the deficiency of copper uptake by cells due to the decreased expression activity of CTR1. The gene discussed is SLC31A1; the disease is familial dilated cardiomyopathy.