In agreement with previous reports, either the effect of the penton and penton-base proteins during initial stages of infection (McIntosh et al., 1971) or the low and transient expression of viral early genes (Rathod et al., 2007) may have been sufficient to induce increased C/EBPβ, C/EBPα and PPARγ mRNAs and lipid accumulation (Figures 3E–G), but only to levels that were comparable to or slightly higher than those induced by MDI in the MK-infected cells (compare Figures 3E, G, I, J with Figures 5A, C, E, F). Here, CEBPB is linked to infection.