Arrest of the cell cycle at Go/G1 phage, morphological changes in cancer cells; activation of apoptosis regulators such as caspaces and Bax protein and p53 and p21 genes; downregulation of transcription factors, such as transcription factor kappa B (NF-kB) and Bcl-2 (B-cell lymphoma 2) gene; and inhibition of enzymes vital for DNA transcription are some examples that confirm the potential of phenolic compounds to accelerate cancer cell death [63,69]. This evidence concerns the gene BCL2 and cancer.