NPPB and myocarditis: The circulating levels of NT-pro BNP and hs-TnI can be differently affected by the mechanisms involved in cardiac dysfunction and/or injury, and the increase of both biomarkers can be a result of powerful stressor mechanisms which cause relevant alterations of cardiac function (resulting in increased natriuretic peptides), significant damage of cardiomyocytes and myocarditis, or perhaps direct effects of SARS-CoV2 on microvasculature (resulting in increased troponins) [7,37,38].