Its virus infection ability is closely related to the increased expression of the activation transcription factor 6 (ATF6), a key regulator of lipid metabolism, and CUR treatment can significantly inhibit the expression of fatty acid synthesis FASN, and finally reduce the synthesis of ATF6, which suggested that CUR might suppress the duplication of CSF virus by regulating lipids metabolism [81]. The gene discussed is ATF6; the disease is viral infectious disease.