The global expression profile enriched critical pathways already related to the endometriosis condition, such as PI3K signaling via AKT to mTORC1 [63,64,65], mTORC1 signaling [97], TNFA signaling via NFkB [76,77], IL6 STAT3 signaling [75], TGF beta signaling [72,73], and hypoxia via HIF1A targets [74]. Here, NFKB1 is linked to endometriosis.