Furthermore, ATF3 and NR4A1 play a role in the process of endometrial decidualization and in the epithelial–mesenchymal transition [45,57], while EGR1, ZFP36, ID1, and ID3 play an important role in the processes of inflammation associated with endometriosis as well as the ability to interact with TNFA and hypoxia [58,59]. Here, EGR1 is linked to endometriosis.