A role for ZnT2 in clearance and tissue repair is consistent with observations of increased neutrophils and dead cells in the colon of ZnT2-null mice during the phase of early resolution; however, by day 21 these differences were modest and further mechanistic studies are required to both understand the molecular role of ZnT2 autophagy, as well as its role in successful resolution of infectious colitis. This evidence concerns the gene SLC30A2 and infectious colitis.