In relation to fibrosis, miR-199a-3p was reported to suppress cytokine signalling-7 (SOCS7) to upregulate signal transducer and activate transcription 3 (STAT3) activation, which is directly induced by TGF- β-driven p53 upregulation in renal fibrosis [35]. The gene discussed is TP53; the disease is renal fibrosis.