Some in vitro studies indicate that AG enhances lipogenesis (and NAFLD), activating GHS-R on hepatocytes and leading to an increase in triglyceride (TG) synthesis [40], while other studies indicate a decrease in TG accumulation with concomitant increases in glutathione peroxidase (GPx) in high-fat diet (HFD)-induced NAFLD and AG treatment [41]. Here, GHSR is linked to metabolic dysfunction-associated steatotic liver disease.