Additionally, to the cDC2/moDC (CD1c+CD14+) population, Michea et al., has identified the prevalent infiltration in breast cancer of CD1c−CD14+ cells with a monocytes/macrophage- like phenotype and CD56+CD14+ cells, with interferon-producing potentiality whose nature is still controversial [165]. This evidence concerns the gene CD1C and breast carcinoma.