Initially, Comincini et al. [45] identified miR-17-5p as a modulator of different autophagy-related proteins (ATGs), demonstrating that anti-miR-17-5p administration results in an increase in MAP1LC3B (microtubule-associated protein 1 light chain 3 beta) and ATG7 (autophagy-related 7) protein expression, and subsequently, yields an activation of autophagy through autophagosome formation in glioblastoma T98G cells. Here, ATG7 is linked to glioblastoma.