EIF3J and gastric cancer: For instance, GC patients have remarkably elevated levels of LINC01503, which mediates cell cycle progression and tumorigenesis [19]; SNHG22 promotes the proliferation and invasion of GC [20]; patients with chemotherapy-resistant GC express high EIF3J-DT levels [21]; meanwhile, SEMA3B-AS1 is downregulated in gastric cardia adenocarcinoma and suppresses tumor progression [22].