Our results showed that AML and CML differ in the expression of GBF1, CD177, OLFM4 and peptidylprolyl isomerase B (PPIB) implicated in the intercellular interactions and chemotaxis regulation to regulate tumor cell migration and invasion [67,85,86,91,92,93,94,95]. The gene discussed is CD177; the disease is acute myeloid leukemia.