BCL2 and neoplasm: TAX promoted the expression of the pro-apoptotic molecules such as Bax, Caspase-3, Caspase-9, Cyt-C and inhibited the expression of the anti-apoptosis molecules such as Bcl-2 and PARP1, indicating that TAX efficiently induced apoptosis in tumor cells in mRNA or protein (Figure 9A–C) levels.