It showed that TAX downregulated the expression of mesenchymal markers N-cadherin, vimentin, snail1, snail2, MMP9 and upregulated the expression of the epithelial marker E-cadherin in a dose-dependent manner in mRNA and protein levels (Figure 2B–D), suggesting that TAX efficiently inhibited the EMT process, thereby inhibiting the migration of lung cancer cells. Here, SNAI2 is linked to lung cancer.