It showed that TAX downregulated the expression of mesenchymal markers N-cadherin, vimentin, snail1, snail2, MMP9 and upregulated the expression of the epithelial marker E-cadherin in a dose-dependent manner in mRNA and protein levels (Figure 2B–D), suggesting that TAX efficiently inhibited the EMT process, thereby inhibiting the migration of lung cancer cells. The gene discussed is SNAI1; the disease is lung carcinoma.