Dysregulation of human genes via HPV integration may also confer cancer-promoting properties; indeed, activating HPV integrations near cervical cancer oncogenes (e.g., TP63 and MYC) and tumour suppressors (e.g., RAD51) have been observed, indicating that integration can both amplify and disrupt human gene expression [21,43,191,192]. The gene discussed is MYC; the disease is cancer.