We recently demonstrated that ZIKV infection in neonatal mice not only resulted in abnormal ECG with prolonged PR interval (biomarker of atrioventricular block), widened QRS (biomarker of intraventricular block), and elevated ST wave (biomarker of impairment of myocardium), implying defects of the myocardial functions, but also caused increased levels of CCL2, CXCL9, and CXCL10 that are inducers of cardiovascular diseases and the infarction-like myocardial pathogenesis (data not shown). The gene discussed is CXCL9; the disease is Zika virus infectious disease.