Generally, these results indicated that apoptosis and pyroptosis played an important role in RSV-infected asthma mice and LPS-mediated 16HBE cells, and CAD potential inhibits the TLR3/NLRP3/NF-κB/IRF3 signaling pathway and subsequently suppresses airway inflammation and pyroptosis. The gene discussed is NLRP3; the disease is asthma.