MYC and neoplasm: Cluster1 was significantly enriched in the estrogen response early and late pathways and the stromal activation-related TGF β pathways. However, Cluster2 was significantly enriched in tumor immune escape- and carcinogenesis-related pathways, such as the glycolysis, ROS, mTORC1, Hedgehog, Wnt/β-catenin, Myc, and inflammatory response pathways (Figure 3A and Table S4).