Zhou et al. [108] found that EA can improve neuronal damage and regulate synaptic plasticity in rats with cerebral ischemia, and that its improvement mechanism is related to downregulation of miR-191a-5p, targeting of neuronal calcium sensor 1 (NCS-1), BDNF, and GAP43, and protection of neurons. This evidence concerns the gene NCS1 and Cerebral ischemia.