Past studies have unveiled various mechanisms implicated in the aforementioned dietary regimens, such as overall energy expenditure reduction, antioxidant supplementation, increased production of hepatokines (e.g., FGF21) (67–71), etc. Due to the scarcity of relevant literature on AAA, we will focus on two emerging dietary mechanisms that are recently implicated in AAA pathophysiologies while more in-depth investigations are still needed. Here, FGF21 is linked to triple-A syndrome.