PARP1 and neoplasm: showed the effects of the molar activity on the uptake of a PARP inhibitor-derived compound, [131I]-I2-PARPi, in tumour and other organs, whereby lower molar activity of [131I]-I2-PARPi (0.185 GBq/μmol) resulted in higher uptake in tumour and other organs, compared to the higher molar activity of [131I]-I2-PARPi (9.25 GBq/μmol) [15].