MAPK9 and cardiac hypertrophy: In the present study, we found that ECH down‐regulated phosphorylation levels of SAPK/JNK, the activation of JNK mediated cardiac hypertrophy, apoptosis, interstitial fibrosis, remodelling and dysfunction and inhibited the activation of JNK can protect against cardiac remodelling.30, 31, 32, 33, 34