Decreasing N-cadherin expression in glioblastoma cells has been shown to impair their focal adhesion and enhance their migratory capacity [55], and an in vitro study of glioblastoma autophagy has been shown to facilitate the degradation of SNAIL family proteins leading to upregulation of the N-cadherin level, which suggests that autophagy may suppress the invasive properties of glioblastomas [56, 57]. Here, SNAI1 is linked to glioblastoma.