It has been shown that TRPA1 can be activated by a number of noxious chemical agents as well as by the inflammatory peptide bradykinin (BK).3 BK has for a long time been implicated in the inflammatory disease acute pancreatitis4 and since TRPA1 is expressed widely in the gastro-intestinal tract1, it would be of interest to investigate the possible role of this channel in the pathophysiology of pancreatitis. This evidence concerns the gene TRPA1 and pancreatitis.