NFKB1 and endometriosis: Some previous studies have shown that the NF-κB signaling pathway was activated in the eutopic secretory endometrium of endometriosis patients, which was also observed in our study, indicating that the absence of decreased p65 activity in the secretory endometrium could participate in endometrial biologic alterations during the implantation window in endometriosis patients [37–39].