As shown in Figures 7(a)–7(h), the “ribosome,” “intestinal immune network for IgA production,” “systemic lupus erythematosus,” “asthma,” “nod like receptor signaling,” “glycosaminoglycan degradation,” “viral myocarditis,” and “cell adhesion molecules cams” pathways were found to be significantly enriched in the poor-prognosis group. This evidence concerns the gene CD79A and asthma.